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Developmental Studies Hybridoma Bank mhc iix
Surviving Spin1 M5 mice exhibit major defects in soleus, tibialis anterior, and diaphragm. ( a,b ) Appearance ( a ) and average body weight ( b ) of Spin1 M5 and control mice ( n =5 females in each category) at 30 weeks of age. Error bars represent +S.D., ** P <0.01. ( c ) Degeneration of the soleus (SOL) muscle in adult Spin1 M5 compared with control mice exemplified at 16 weeks of age. ( d ) Hind limb muscles (gastrocnemius (GC), plantaris (PL), soleus, tibialis anterior (TA), extensor digitorum longus (EDL), and quadriceps (QC)) of Spin1 M5 and control mice at 30 weeks of age. Arrows point at the soleus embedded in gastrocnemius and plantaris, which is visible in control but degenerated in Spin1 M5 mice. ( e ) Hematoxylin & eosin (H&E) staining of gastrocnemius, soleus, TA, and EDL muscle of Spin1 M5 and control mice at 30 weeks of age. ( f ) Fiber types in glycolytic (white) or oxidative (red) parts of the TA of Spin1 M5 and control mice at 15 weeks of age (top and middle rows) observed by immunofluorescence (IF) staining. Tissue sections were stained with selective antibody directed against <t>MHC-I</t> (purple), MHC-IIb <t>(cyan),</t> <t>MHC-IIa</t> (red), and MHC-IIx (green). For comparison, NADH staining was included (bottom row). Fibers with abnormal NADH staining are marked with arrows. Corresponding fibers in each column of images are squared
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Sophia Genetics alamut batch version 1 11
Surviving Spin1 M5 mice exhibit major defects in soleus, tibialis anterior, and diaphragm. ( a,b ) Appearance ( a ) and average body weight ( b ) of Spin1 M5 and control mice ( n =5 females in each category) at 30 weeks of age. Error bars represent +S.D., ** P <0.01. ( c ) Degeneration of the soleus (SOL) muscle in adult Spin1 M5 compared with control mice exemplified at 16 weeks of age. ( d ) Hind limb muscles (gastrocnemius (GC), plantaris (PL), soleus, tibialis anterior (TA), extensor digitorum longus (EDL), and quadriceps (QC)) of Spin1 M5 and control mice at 30 weeks of age. Arrows point at the soleus embedded in gastrocnemius and plantaris, which is visible in control but degenerated in Spin1 M5 mice. ( e ) Hematoxylin & eosin (H&E) staining of gastrocnemius, soleus, TA, and EDL muscle of Spin1 M5 and control mice at 30 weeks of age. ( f ) Fiber types in glycolytic (white) or oxidative (red) parts of the TA of Spin1 M5 and control mice at 15 weeks of age (top and middle rows) observed by immunofluorescence (IF) staining. Tissue sections were stained with selective antibody directed against <t>MHC-I</t> (purple), MHC-IIb <t>(cyan),</t> <t>MHC-IIa</t> (red), and MHC-IIx (green). For comparison, NADH staining was included (bottom row). Fibers with abnormal NADH staining are marked with arrows. Corresponding fibers in each column of images are squared
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Fisher Scientific fisher scientific process 11 twin-screw extruder
Surviving Spin1 M5 mice exhibit major defects in soleus, tibialis anterior, and diaphragm. ( a,b ) Appearance ( a ) and average body weight ( b ) of Spin1 M5 and control mice ( n =5 females in each category) at 30 weeks of age. Error bars represent +S.D., ** P <0.01. ( c ) Degeneration of the soleus (SOL) muscle in adult Spin1 M5 compared with control mice exemplified at 16 weeks of age. ( d ) Hind limb muscles (gastrocnemius (GC), plantaris (PL), soleus, tibialis anterior (TA), extensor digitorum longus (EDL), and quadriceps (QC)) of Spin1 M5 and control mice at 30 weeks of age. Arrows point at the soleus embedded in gastrocnemius and plantaris, which is visible in control but degenerated in Spin1 M5 mice. ( e ) Hematoxylin & eosin (H&E) staining of gastrocnemius, soleus, TA, and EDL muscle of Spin1 M5 and control mice at 30 weeks of age. ( f ) Fiber types in glycolytic (white) or oxidative (red) parts of the TA of Spin1 M5 and control mice at 15 weeks of age (top and middle rows) observed by immunofluorescence (IF) staining. Tissue sections were stained with selective antibody directed against <t>MHC-I</t> (purple), MHC-IIb <t>(cyan),</t> <t>MHC-IIa</t> (red), and MHC-IIx (green). For comparison, NADH staining was included (bottom row). Fibers with abnormal NADH staining are marked with arrows. Corresponding fibers in each column of images are squared
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Microgen Inc monospecific anti-viper venom serum (batch number c51, 11.2016, 150ae)
Surviving Spin1 M5 mice exhibit major defects in soleus, tibialis anterior, and diaphragm. ( a,b ) Appearance ( a ) and average body weight ( b ) of Spin1 M5 and control mice ( n =5 females in each category) at 30 weeks of age. Error bars represent +S.D., ** P <0.01. ( c ) Degeneration of the soleus (SOL) muscle in adult Spin1 M5 compared with control mice exemplified at 16 weeks of age. ( d ) Hind limb muscles (gastrocnemius (GC), plantaris (PL), soleus, tibialis anterior (TA), extensor digitorum longus (EDL), and quadriceps (QC)) of Spin1 M5 and control mice at 30 weeks of age. Arrows point at the soleus embedded in gastrocnemius and plantaris, which is visible in control but degenerated in Spin1 M5 mice. ( e ) Hematoxylin & eosin (H&E) staining of gastrocnemius, soleus, TA, and EDL muscle of Spin1 M5 and control mice at 30 weeks of age. ( f ) Fiber types in glycolytic (white) or oxidative (red) parts of the TA of Spin1 M5 and control mice at 15 weeks of age (top and middle rows) observed by immunofluorescence (IF) staining. Tissue sections were stained with selective antibody directed against <t>MHC-I</t> (purple), MHC-IIb <t>(cyan),</t> <t>MHC-IIa</t> (red), and MHC-IIx (green). For comparison, NADH staining was included (bottom row). Fibers with abnormal NADH staining are marked with arrows. Corresponding fibers in each column of images are squared
Monospecific Anti Viper Venom Serum (Batch Number C51, 11.2016, 150ae), supplied by Microgen Inc, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Surviving Spin1 M5 mice exhibit major defects in soleus, tibialis anterior, and diaphragm. ( a,b ) Appearance ( a ) and average body weight ( b ) of Spin1 M5 and control mice ( n =5 females in each category) at 30 weeks of age. Error bars represent +S.D., ** P <0.01. ( c ) Degeneration of the soleus (SOL) muscle in adult Spin1 M5 compared with control mice exemplified at 16 weeks of age. ( d ) Hind limb muscles (gastrocnemius (GC), plantaris (PL), soleus, tibialis anterior (TA), extensor digitorum longus (EDL), and quadriceps (QC)) of Spin1 M5 and control mice at 30 weeks of age. Arrows point at the soleus embedded in gastrocnemius and plantaris, which is visible in control but degenerated in Spin1 M5 mice. ( e ) Hematoxylin & eosin (H&E) staining of gastrocnemius, soleus, TA, and EDL muscle of Spin1 M5 and control mice at 30 weeks of age. ( f ) Fiber types in glycolytic (white) or oxidative (red) parts of the TA of Spin1 M5 and control mice at 15 weeks of age (top and middle rows) observed by immunofluorescence (IF) staining. Tissue sections were stained with selective antibody directed against <t>MHC-I</t> (purple), MHC-IIb <t>(cyan),</t> <t>MHC-IIa</t> (red), and MHC-IIx (green). For comparison, NADH staining was included (bottom row). Fibers with abnormal NADH staining are marked with arrows. Corresponding fibers in each column of images are squared
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Raiffeisen Waren chicken feed diet k (11 4) o.k
Surviving Spin1 M5 mice exhibit major defects in soleus, tibialis anterior, and diaphragm. ( a,b ) Appearance ( a ) and average body weight ( b ) of Spin1 M5 and control mice ( n =5 females in each category) at 30 weeks of age. Error bars represent +S.D., ** P <0.01. ( c ) Degeneration of the soleus (SOL) muscle in adult Spin1 M5 compared with control mice exemplified at 16 weeks of age. ( d ) Hind limb muscles (gastrocnemius (GC), plantaris (PL), soleus, tibialis anterior (TA), extensor digitorum longus (EDL), and quadriceps (QC)) of Spin1 M5 and control mice at 30 weeks of age. Arrows point at the soleus embedded in gastrocnemius and plantaris, which is visible in control but degenerated in Spin1 M5 mice. ( e ) Hematoxylin & eosin (H&E) staining of gastrocnemius, soleus, TA, and EDL muscle of Spin1 M5 and control mice at 30 weeks of age. ( f ) Fiber types in glycolytic (white) or oxidative (red) parts of the TA of Spin1 M5 and control mice at 15 weeks of age (top and middle rows) observed by immunofluorescence (IF) staining. Tissue sections were stained with selective antibody directed against <t>MHC-I</t> (purple), MHC-IIb <t>(cyan),</t> <t>MHC-IIa</t> (red), and MHC-IIx (green). For comparison, NADH staining was included (bottom row). Fibers with abnormal NADH staining are marked with arrows. Corresponding fibers in each column of images are squared
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Eppendorf AG speedvac
Surviving Spin1 M5 mice exhibit major defects in soleus, tibialis anterior, and diaphragm. ( a,b ) Appearance ( a ) and average body weight ( b ) of Spin1 M5 and control mice ( n =5 females in each category) at 30 weeks of age. Error bars represent +S.D., ** P <0.01. ( c ) Degeneration of the soleus (SOL) muscle in adult Spin1 M5 compared with control mice exemplified at 16 weeks of age. ( d ) Hind limb muscles (gastrocnemius (GC), plantaris (PL), soleus, tibialis anterior (TA), extensor digitorum longus (EDL), and quadriceps (QC)) of Spin1 M5 and control mice at 30 weeks of age. Arrows point at the soleus embedded in gastrocnemius and plantaris, which is visible in control but degenerated in Spin1 M5 mice. ( e ) Hematoxylin & eosin (H&E) staining of gastrocnemius, soleus, TA, and EDL muscle of Spin1 M5 and control mice at 30 weeks of age. ( f ) Fiber types in glycolytic (white) or oxidative (red) parts of the TA of Spin1 M5 and control mice at 15 weeks of age (top and middle rows) observed by immunofluorescence (IF) staining. Tissue sections were stained with selective antibody directed against <t>MHC-I</t> (purple), MHC-IIb <t>(cyan),</t> <t>MHC-IIa</t> (red), and MHC-IIx (green). For comparison, NADH staining was included (bottom row). Fibers with abnormal NADH staining are marked with arrows. Corresponding fibers in each column of images are squared
Speedvac, supplied by Eppendorf AG, used in various techniques. Bioz Stars score: 99/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Lakeshore Biomaterials poly(d,l-lactide-co-glicolide) (plga)
Surviving Spin1 M5 mice exhibit major defects in soleus, tibialis anterior, and diaphragm. ( a,b ) Appearance ( a ) and average body weight ( b ) of Spin1 M5 and control mice ( n =5 females in each category) at 30 weeks of age. Error bars represent +S.D., ** P <0.01. ( c ) Degeneration of the soleus (SOL) muscle in adult Spin1 M5 compared with control mice exemplified at 16 weeks of age. ( d ) Hind limb muscles (gastrocnemius (GC), plantaris (PL), soleus, tibialis anterior (TA), extensor digitorum longus (EDL), and quadriceps (QC)) of Spin1 M5 and control mice at 30 weeks of age. Arrows point at the soleus embedded in gastrocnemius and plantaris, which is visible in control but degenerated in Spin1 M5 mice. ( e ) Hematoxylin & eosin (H&E) staining of gastrocnemius, soleus, TA, and EDL muscle of Spin1 M5 and control mice at 30 weeks of age. ( f ) Fiber types in glycolytic (white) or oxidative (red) parts of the TA of Spin1 M5 and control mice at 15 weeks of age (top and middle rows) observed by immunofluorescence (IF) staining. Tissue sections were stained with selective antibody directed against <t>MHC-I</t> (purple), MHC-IIb <t>(cyan),</t> <t>MHC-IIa</t> (red), and MHC-IIx (green). For comparison, NADH staining was included (bottom row). Fibers with abnormal NADH staining are marked with arrows. Corresponding fibers in each column of images are squared
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Carbolite Gero batch quartz rotary furnace htr 11/150
Surviving Spin1 M5 mice exhibit major defects in soleus, tibialis anterior, and diaphragm. ( a,b ) Appearance ( a ) and average body weight ( b ) of Spin1 M5 and control mice ( n =5 females in each category) at 30 weeks of age. Error bars represent +S.D., ** P <0.01. ( c ) Degeneration of the soleus (SOL) muscle in adult Spin1 M5 compared with control mice exemplified at 16 weeks of age. ( d ) Hind limb muscles (gastrocnemius (GC), plantaris (PL), soleus, tibialis anterior (TA), extensor digitorum longus (EDL), and quadriceps (QC)) of Spin1 M5 and control mice at 30 weeks of age. Arrows point at the soleus embedded in gastrocnemius and plantaris, which is visible in control but degenerated in Spin1 M5 mice. ( e ) Hematoxylin & eosin (H&E) staining of gastrocnemius, soleus, TA, and EDL muscle of Spin1 M5 and control mice at 30 weeks of age. ( f ) Fiber types in glycolytic (white) or oxidative (red) parts of the TA of Spin1 M5 and control mice at 15 weeks of age (top and middle rows) observed by immunofluorescence (IF) staining. Tissue sections were stained with selective antibody directed against <t>MHC-I</t> (purple), MHC-IIb <t>(cyan),</t> <t>MHC-IIa</t> (red), and MHC-IIx (green). For comparison, NADH staining was included (bottom row). Fibers with abnormal NADH staining are marked with arrows. Corresponding fibers in each column of images are squared
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Surviving Spin1 M5 mice exhibit major defects in soleus, tibialis anterior, and diaphragm. ( a,b ) Appearance ( a ) and average body weight ( b ) of Spin1 M5 and control mice ( n =5 females in each category) at 30 weeks of age. Error bars represent +S.D., ** P <0.01. ( c ) Degeneration of the soleus (SOL) muscle in adult Spin1 M5 compared with control mice exemplified at 16 weeks of age. ( d ) Hind limb muscles (gastrocnemius (GC), plantaris (PL), soleus, tibialis anterior (TA), extensor digitorum longus (EDL), and quadriceps (QC)) of Spin1 M5 and control mice at 30 weeks of age. Arrows point at the soleus embedded in gastrocnemius and plantaris, which is visible in control but degenerated in Spin1 M5 mice. ( e ) Hematoxylin & eosin (H&E) staining of gastrocnemius, soleus, TA, and EDL muscle of Spin1 M5 and control mice at 30 weeks of age. ( f ) Fiber types in glycolytic (white) or oxidative (red) parts of the TA of Spin1 M5 and control mice at 15 weeks of age (top and middle rows) observed by immunofluorescence (IF) staining. Tissue sections were stained with selective antibody directed against <t>MHC-I</t> (purple), MHC-IIb <t>(cyan),</t> <t>MHC-IIa</t> (red), and MHC-IIx (green). For comparison, NADH staining was included (bottom row). Fibers with abnormal NADH staining are marked with arrows. Corresponding fibers in each column of images are squared
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Qingdao Growful Pharmaceutical Co Ltd jnw
Surviving Spin1 M5 mice exhibit major defects in soleus, tibialis anterior, and diaphragm. ( a,b ) Appearance ( a ) and average body weight ( b ) of Spin1 M5 and control mice ( n =5 females in each category) at 30 weeks of age. Error bars represent +S.D., ** P <0.01. ( c ) Degeneration of the soleus (SOL) muscle in adult Spin1 M5 compared with control mice exemplified at 16 weeks of age. ( d ) Hind limb muscles (gastrocnemius (GC), plantaris (PL), soleus, tibialis anterior (TA), extensor digitorum longus (EDL), and quadriceps (QC)) of Spin1 M5 and control mice at 30 weeks of age. Arrows point at the soleus embedded in gastrocnemius and plantaris, which is visible in control but degenerated in Spin1 M5 mice. ( e ) Hematoxylin & eosin (H&E) staining of gastrocnemius, soleus, TA, and EDL muscle of Spin1 M5 and control mice at 30 weeks of age. ( f ) Fiber types in glycolytic (white) or oxidative (red) parts of the TA of Spin1 M5 and control mice at 15 weeks of age (top and middle rows) observed by immunofluorescence (IF) staining. Tissue sections were stained with selective antibody directed against <t>MHC-I</t> (purple), MHC-IIb <t>(cyan),</t> <t>MHC-IIa</t> (red), and MHC-IIx (green). For comparison, NADH staining was included (bottom row). Fibers with abnormal NADH staining are marked with arrows. Corresponding fibers in each column of images are squared
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Purolite Life Sciences ct275dr
Condensation of Acetone to Mesitylene
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Image Search Results


Surviving Spin1 M5 mice exhibit major defects in soleus, tibialis anterior, and diaphragm. ( a,b ) Appearance ( a ) and average body weight ( b ) of Spin1 M5 and control mice ( n =5 females in each category) at 30 weeks of age. Error bars represent +S.D., ** P <0.01. ( c ) Degeneration of the soleus (SOL) muscle in adult Spin1 M5 compared with control mice exemplified at 16 weeks of age. ( d ) Hind limb muscles (gastrocnemius (GC), plantaris (PL), soleus, tibialis anterior (TA), extensor digitorum longus (EDL), and quadriceps (QC)) of Spin1 M5 and control mice at 30 weeks of age. Arrows point at the soleus embedded in gastrocnemius and plantaris, which is visible in control but degenerated in Spin1 M5 mice. ( e ) Hematoxylin & eosin (H&E) staining of gastrocnemius, soleus, TA, and EDL muscle of Spin1 M5 and control mice at 30 weeks of age. ( f ) Fiber types in glycolytic (white) or oxidative (red) parts of the TA of Spin1 M5 and control mice at 15 weeks of age (top and middle rows) observed by immunofluorescence (IF) staining. Tissue sections were stained with selective antibody directed against MHC-I (purple), MHC-IIb (cyan), MHC-IIa (red), and MHC-IIx (green). For comparison, NADH staining was included (bottom row). Fibers with abnormal NADH staining are marked with arrows. Corresponding fibers in each column of images are squared

Journal: Cell Death & Disease

Article Title: The histone code reader Spin1 controls skeletal muscle development

doi: 10.1038/cddis.2017.468

Figure Lengend Snippet: Surviving Spin1 M5 mice exhibit major defects in soleus, tibialis anterior, and diaphragm. ( a,b ) Appearance ( a ) and average body weight ( b ) of Spin1 M5 and control mice ( n =5 females in each category) at 30 weeks of age. Error bars represent +S.D., ** P <0.01. ( c ) Degeneration of the soleus (SOL) muscle in adult Spin1 M5 compared with control mice exemplified at 16 weeks of age. ( d ) Hind limb muscles (gastrocnemius (GC), plantaris (PL), soleus, tibialis anterior (TA), extensor digitorum longus (EDL), and quadriceps (QC)) of Spin1 M5 and control mice at 30 weeks of age. Arrows point at the soleus embedded in gastrocnemius and plantaris, which is visible in control but degenerated in Spin1 M5 mice. ( e ) Hematoxylin & eosin (H&E) staining of gastrocnemius, soleus, TA, and EDL muscle of Spin1 M5 and control mice at 30 weeks of age. ( f ) Fiber types in glycolytic (white) or oxidative (red) parts of the TA of Spin1 M5 and control mice at 15 weeks of age (top and middle rows) observed by immunofluorescence (IF) staining. Tissue sections were stained with selective antibody directed against MHC-I (purple), MHC-IIb (cyan), MHC-IIa (red), and MHC-IIx (green). For comparison, NADH staining was included (bottom row). Fibers with abnormal NADH staining are marked with arrows. Corresponding fibers in each column of images are squared

Article Snippet: For immunofluorescence staining the following primary antibodies were used: SPIN1(5865) 1 μ g/ml; Pax7 (PAX7, DSHB, batch 7/2/15) 2 μ g/ml; Tcf4 (6H5-3, Millipore, Darmstadt, Germany, 05-511, batch 2155406) 10 μ g/ml; Ankrd1 (Proteintech Group, Manchester, UK, 11427-1-AP, batch 1951) 1:100; Ankrd2 (Proteintech Group, 11821-1-AP, batch 7649) 1:100; dystrophin (Abcam, Cambridge, UK, ab15277, batch GR226781-6) 1:500; MHC-I (NOQ7.5.4D, Sigma, Munich, Germany, M8421, batch 035M4792V) 1:2000; MHC-IIa (SC-71, DSHB, batch 4/7/16) 1:10; MHC-IIx (6H1, DSHB, batch 3/3/16) 1:6; MHC-IIb (BF-F3, DSHB, batch 5/12/16) 1:20; MCH, skeletal, fast (MY-32, Sigma, M4276, batch 083M4790V) 1:1000; GNZ (anti-GFP, Abcam, ab13970, batch GR236651) 1:1000; normal rabbit IgG (Santa Cruz, Heidelberg, Germany, sc-2027).

Techniques: Control, Muscles, Staining, Immunofluorescence, Comparison

Condensation of Acetone to Mesitylene

Journal: Chemical Reviews

Article Title: Benzenoid Aromatics from Renewable Resources

doi: 10.1021/acs.chemrev.4c00087

Figure Lengend Snippet: Condensation of Acetone to Mesitylene

Article Snippet: 11 , 130 , 25 , Batch , 6 h , Purolite CT275DR , , , 12.5 , ( ) .

Techniques: